Question ID: WS-79
Submitted by: Society for Immunotherapy of Cancer
March 18, 2011
Recent work has suggested that standard cancer therapeutics (chemotherapy, radiation therapy, tumor-targeting monoclonal antibodies) all are capable of facilitating a host immune response against the tumor. Mechanistic studies in mice have indicated that the immune response is in fact required for a maximal therapeutic effect of these agents. These data raise the critical question, “Are all effective cancer therapies working, at least in part, through eliciting an anti-tumor immune response?” This is not so far-fetched, as we have known for some time that anti-bacterial antibiotics do not completely cure infections in neutropenic patients until the neutrophil count recovers.
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